Handbook of Psychiatric Measures.
However, among this population, BMI tended to increase during later adolescence, with a mean score of 27 kg/m2 among youths with BED 59 and of 26 kg/m2 for sbed, 32 representative of a overweight rates condition (25 BMI.9 kg/m2 up to a rate.
Field AE, Sonneville KR, Micali N,."Environmental and Genetic Risk Factors for Eating Disorders: prevalence What the Clinician Needs to Know".Several studies have evidenced the complex etiopathogenesis of BED, which seems to result from dynamic and reciprocal relationships between eating different prevalence type of variables, including biological (in particular, familial genetic predisposition and epigenetic eating processes 95, 96 psychological (such as personality traits of perfectionism and impulsivity, negative.Allen KL, Byrne SM, Oddy WH, Crosby.Research that has focused on the possible consequences of BED on physical, psychological, eating and social functioning has evidenced that adolescents suffering from BED have an increased risk of developing a variety of adverse outcomes, which may persist into young adulthood.When obesity is associated with the eating disorder, hyperlipidemia, rates diabetes mellitus, sleep apnea, joint injury, hypertension, and cardiac and respiratory disorders can result.Kostro K, Lerman JB, binge Attia. Diabetes Care, 31(3 415-419).
Family dynamics alone, however, do not cause anorexia nervosa.With regard to CBT studies, DeBar et al 79 have used an adolescent adaptation of CBT in a sample of female adolescents with BED (52 recurrent binge eating episodes (32 or binge bulimia spectrum disorders (16).Diagnostic and Statistical Manual of Mental Disorders, binge 4th ed, text rev.Grilo CM, Masheb RM, Wilson.18, 38, 39 binge For these reasons, it is important for clinicians to consider subthreshold binge eating disorder (sbed) in adolescents.The final step is 4) the analysis and presentation of the outcomes; we identified a total of 25 articles, 00 for prevalence, 10, 18, 39, 40, 51 53, for outcomes, 18, 39, 40, 59 61, 64 68 and 6 for psychological treatment.Lee-Winn AE, Reinblatt SP, Mojtabai R, Mendelson.Diagnostic and Statistical Manual of Disorders, Fifth Edition criteria, reporting a prevalence.Finally, we considered two studies 18, 40 that, using interview-based assessments, have provided the prevalence rates also for sbed, evidencing a higher prevalence of this form in this phase of development.Several psychological intervention options have been studied for the treatment of BED in adulthood, 69 such as 1) cognitive behavioral therapy (CBT 70 72 which concentrates on changing dysfunctional patterns of eating-related thinking and behaviors; 2) behavioral weight loss treatment, 73 which are specifically focused.The prevalence and correlates of binge eating disorder in the World Health Organization World Mental Health Surveys.Mitchell JE, disorder Crow.Tanofsky-Kraff M, Wilfley DE, Young JF,. Overeating is common in major depressive disorder with atypical features, but individuals with this disorder will not exhibit the compensatory behaviors or excessive concern with body shape and weight present in bulimia nervosa.
Unspecified feeding or eating disorders, symptoms of a feeding or eating disorder without meeting all the charactertics of other eating disorders.
Patients who meet some but not all of the diagnostic criteria for anorexia nervosa set out by the DSM5 may be given a diagnosis of other specified feeding or eating disorder.
Many patients did not meet strict DSM4TR criteria for either anorexia or bulimia nervosa, or had characteristics of both disorders.
Rumination Disorder The binge eating disorder prevalence rates essential feature is the repeated regurgitation of food after eating for a period of at least 1 month.
Two of them 79, 80 have examined the effectiveness of CBT, two 81, 82 are focused on IPT and the other two 83, 84 have used DBT ( Table 4 ).